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epetitive
micro-traumata (injuries), unresolved single injury, inflammation, poor
posture or maladaptive movement habits at play or at work, stress, lack
of sleep, or any combination of the above will produce tightness or abnormal
contraction of skeletal muscles. The investing fascia becomes taut and
bound down. Circulation to and from the muscles is decreased, resulting
in the accumulation of the end-products of muscle metabolism, particularly
lactic acid and potassium ions. Localized areas of muscle tenderness called
TRIGGER POINTS are formed. These are extremely sensitive and will
fire impulses (under the slightest provocation such as pressure and stretching),
to distant tissues, producing pain and consequent loss of motion at remote
locations. This causes the further accumulation of muscle toxins, more
muscle and fascial tightness, more pain, etc., perpetuating the MYOFASCIAL
PAIN cycle.
This is an extremely common cause of pain anywhere in the
body: head, face, neck, shoulders, chest, arms, low back, buttocks, legs,
feet, etc. To understand more about this ubiquitous problem, a
few definitions and explanations are in order:
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Fascia is a tough connective tissue lining, covering
and investing muscles, and, indeed, all cells, tissues and organs.
Fascia is three-dimensional and is continuous throughout the body.
Anything affecting fascia in one area is manifested to some extent
in all body regions.
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Pain is an abnormal, unpleasant EMOTIONAL and sensory
experience caused by actual or perceived injury. This results in the
stimulation of nerve endings called nociceptors. These impulses are
transmitted to the spinal cord and then to the brain where they register
as pain.
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Trigger points (TrP) are foci of hyperirritability
in muscle, fascia or ligaments (connecting bone to bone as in joints).
They are characterized by taut fibrous bands, a twitch response when
stimulated, and constant areas of referred pain. The pain patterns
thus produced are called myofascial pain syndromes. There are
several types and locations of trigger points:
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Active TrPs are always tender. They prevent full
lengthening of the muscle and weaken it. Direct compression, stretching,
or other sources of irritation such as accumulation of the toxic chemical
products of muscle metabolism or lack of oxygen will ignite the TrP.
From it, localized pain is produced in a specific area with associated
autonomic changes. These may include increased or reduced skin temperature,
sweating or dryness. The area of referred pain is often distant from
the TrP.
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Latent TrPs may not be painful to pressure, but
result in muscle weakness and restricted motion. There are also secondary
and satellite TrPs, the explanation of which is beyond the
scope of this article.
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Ligamentous TrPs are found in lax, stretched ligaments
as a result of aging, trauma and/or poor posture, particularly those
ligaments involved in the support of the axial (vertebral column and
pelvis) or appendicular skeleton.
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Periosteal TrPs are found on the surface of bone
usually at the site of ligament or tendon attachment and related to
tension on that area from stretched ligaments.
PERPETUATING FACTORS
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Alignment Or Postural Factors: Gravity in combination
with aging, long term poor posture, and/or repeated injury causes
laxity of the axial (trunk and pelvis) and appendicular skeletal ligaments.
This is most relevant in the spine and pelvis. Tightness of the psoas
major muscle combined with weakness of the abdominal muscles, particularly
the pelvic attachment of the obliques and recti, combine to perpetuate
a downward tilting of the pelvis and an increase in the lumbar lordosis.
Thus, the ligaments connecting the pelvis to the vertebral column
and to the lower extremities are stretched and their nociceptors depolarized,
initiating the pain cycle. The same applies to the pelvic and low
back muscles which now are tightened and ischemic.
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Other Perpetuating Factors: include leg length
disparity or pelvic tilt; hyperpronation (inward rolling of the foot);
nutritional, metabolic, endocrine, postural and emotional factors
as well as bacterial or viral infections or parasitic infestations.
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Major Myofascial Pain Syndromes: The trigger points
I have found most frequently related to pain complaints in our practice
are located in the following muscles: iliopsoas, quadratus lumborum,
gluteus medius, gluteus minimus, piriformis, hamstrings, trapezius,
levator scapula, scalenus anticus.
TREATMENT
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Passive stretching can be dangerous: "no pain no gain"
is a rule with strict limitations. The inadequately trained person
stretching you has no conception of your pain or tolerance. Stretch
yourself or let a trained therapist help.
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The presence of a TrP in a muscle can be suspected when
stretching or attempting to strengthen a muscle group is either
fruitless or results in aggravation of pain with the pattern being
repetitive. Don’t attempt to stretch or strengthen a muscle with
TrPs.
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Any discomfort while stretching should be experienced
in the belly of the muscle, NOT at the point of attachment to bone
or in the tendon.
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Remember: not everybody needs to stretch and not every
muscle needs to be stretched. Older people with extremely lax ligaments
may depend on hypertonic muscles to support unstable skeletal structures
and may react poorly to attempts to stretch.
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Treatment of myofascial pain syndromes consists principally
of the following measures and is part of the program available at
Catskill Rehabilitation & Sports Medicine:
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Identify and correct all possible perpetuating factors.
Of particular importance are: correction of postural imbalances with
short leg lifts; correction of faulty foot mechanics with orthotics.
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Identify and treat all trigger points with ultra sound
and low volt electrical stimulation, dry needling, injection with
a local anaesthetic, acupuncture, spray and stretch with a topical
local anaesthetic or any combination of the above.
- Trigger point injections, massage, soft tissue mobilization.
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Re-establish normal, restorative sleep using muscle relaxants,
acupuncture or a combination of these.
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Pain relief with medication ranging from acetominophen
through non-steroid anti-inflammatories and aspirin to short term
narcotics, if necessary.
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Careful, appropriate stretching and strengthening are
essential components of any successful treatment and rehabilitation
program. Your physical therapist and I will be your guides in such
a program.
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Aerobic exercise, instituted carefully and progressed
gradually to tolerance is vital to recovery and prevention of recurrence.
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